Adolph Mares, MD

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Dr. Adolph Mares is originally from San Antonio, Texas. He received his medical degree from Baylor College of Medicine in Houston, TX, where he also completed his internship and residency in Internal Medicine and a fellowship in Cardiology. Dr. Mares. areas of special interest include nuclear cardiology and echocardiography. He is board certified in Internal Medicine and also in the subspecialty of Cardiovascular Disease.

Specialty:

Clinical Cardiology, Invasive Cardiology, and Echocardiography

Office Location:

Harker Heights

Personal Interests:

Sailing and music

Professional Associations:

• American College of Cardiology
• American College of Physicians
• Texas Medical Association
• Bell County Medical Society
• American Society of Echocardiography

Professional Affiliations:

• Assistant Professor of IM/Cardiology, Texas Tech University Health Science Center, Lubbock, TX (11/99-12/00)
• Assistant Professor of IM/Cardiology, Texas A&M University School of Medicine, Temple, TX (05/97-10/99)
• Assistant Professor of IM/Cardiology, Baylor College of Medicine, Houston, TX (07/94-05/97)

Publications:

• Mares A Jr: The relationship of urinary prostaglandin E2 to urinary volume in hypertensive patients. J Tex Acad Sci Coll Acad 1982;33(1):32-40
• Mares A Jr, Delea C, Bartter FC: The relationship of urinary prostaglandin E2 to urinary volume in hypertensive patients. J Biomed Res Sym 1982;203.
• Davies AO, Mares A, Pool JL, Taylor AA: Mitral valvular prolapse with symptoms of beta-adrenergic hypersensitivity- beta2-adrenergic receptor supercoupling with desensitization on isoproterenol exposure. Am J Med 1987; 82(2):193-201.
• Taylor AA, Davies AO, Mares A, Raschko J, Pool JL, Nelson EB, Mitchell JR:Spectrum of dysautonomia in mitral valvular prolapse. Am J Med 1989; 86(3): 267-274.
• Mares A, Davies AO, Taylor AA: Diversity in supercoupling of B2-adrenergic receptors in orthostatic hypotension. Clin Pharmacol Ther 1990; 47(3):371-381.
• Mares A, Ledbetter SA, Ledbetter DH, Roberts R, Hejtmancik JF: Isolation of human chromosome 14 somatic cell hybrid: Analysis using Alu and Line based PCR. Geromics 1991; 11:215-218.
• Mares A, Towbin J, Bies RD, Roberts R: Molecular biology for the cardiologist. Curr Probl Cardiol 1992; 17(1):1-72.
• Perryman MB, Yu Q-T, Marian AJ, Mares A, Czernuszewicz G, Ifeqwu J, Hill R, Roberts R: Expression of a missense mutation in the mRNA for B-myosin heavy chain in myocardial tissue in hypertrophic cardiomyopathy. J Clin Invest 1992; 90:271-277.
• Marian AJ, Yu Q-T, Mares A, Hill R, Roberts R: Detection of a new mutation in the B-myosin heavy chain gene in a family with hypertrophic cardiomyopathy. J Clin Invest 1992; 909:2156-2165.
• Yu Q-T, Ifegwu J, Marian AJ, Mares A, Hill R, Perryman MB, Bachinski LL, Roberts R: Hypertrophic cardiomyopathy mutation as expressed in messenger RNA of skeletal as well as cardiac muscle. Circulation 1993;87:406-412.
• Marian AJ, Kelly D, Mares A, Fitzgibbons J, Caira T, Yu Q-T, Hill R, Perryman MB, Roberts R: A missense mutation in the B-myosin heavy chain is a predictor of premature sudden cardiac death in patients with hypertrophic cardiomyopathy. J Sports Med Phys Fitness 1994; 34(1):1-10.
• Mares A, Roberts R: The techniques of molecular biology and their application to the cardiomyopathies. Adv. Int Md 1994; 39:395-434.
• Anan R, Greve G, Thierfelder L, Watkins H, McKenna WJ, Solomon S, Vecchio C, Spirito P, Shono H, Nakao S, Tanaka H, Mares A, Towbin JA, Roberts R, Seidman JG, Siedman CE: Prognostic implications of novel B-cardiac myosin heavy chain gene mutations that cause familial cardiomyopathy. J Clin Invest 1994; 93:280-285.

Abstracts:

• Mares A, Davies AO, Taylor AA: Supercoupled beta-adrenergic receptors in patients with mitral valvular prolapse and adrenergic hypersensitivity. Fed Proceed 1985; 44(5):1735 # 7740
• Taylor AA, Davies AO, Mares A, Pool JL, Nelson EB,Mitchell JR: Abnormal beta-receptor function in mitral valvular prolapse patients with hyperadrenergic autonomic dysfunction. Clin Res 1985.
• Mares A, Taylor AA, Davies AO, Pool JL, Nelson EB, Mitchell JR: Adrenergic supersensitivity in patients with orthostatic hypotension. Clin Res 1986;34(2)S:402A
• Davies AO, Mares A: Altered regulation of GTP-ase activity of the guanine nucleotide regulatory protein in patients with mitral valvular prolapse. Clin Res 986; 34(2):397A
• Davies AO, Taylor AA, Mares A, Pool JL, Mitchell JR: Dysautonomia in mitral valvular prolapse: Beta2- adrenergic receptor supersensitivity. Abstracts of the III World Conference of Clinical Pharmacology and Therapeutics, Stockholm, 1986,#106.
• Taylor AA, Davies AO, Mares A, Pool JL, Nelson EB, Mitchell JR: Adrenergic supersensitivity in patients with orthostatic hypotension. Abstracts of the III World Conference of Clinical Pharmoacology and Therapeutics, Stockholm, 1986, #384.
• Mares A, Davies AO, Taylor AA: Conditional regulation by norepinephrine of beta-ardrenergic receptors in patients with mitral valvular prolapse and hyperadrenergic autonomic dysfunction. Clin Res 1987,35:378A.
• Mares A, Ledbetter SA, Ledbetter DH, Perryman MB, Roberts R, Hefmancik JF: Construction of a radiation hybrid map of human chromosome 14 in somatic cell hybrids using polymerase chain reaction. J Cell Biochem 1991;15C:H407.
• Mares A, Hefmancik JF, Ledbetter SA, Ledbetter DH, Perryman MB, Roberts R: Mapping of the chromosome containing the locus for hypertrophic cardiomyopathy using polymerase chain reaction and radiation hybrids. J Am Coll Cardiol 1991; 17(2):165A.
• Mares A, Ledbetter DH, Ledbetter SA, Perryman MB, Donis-Keller H, Roberts R, Hejtmancik JF: Radiation hybrids from chromosome 14: IRS-PCR characterization and initial mapping studies: Amer J Hum Genet 1991;49(4):384.
• Perryman MB, Mares A, Hejtmancik JF, Gooch G, Roberts R: The beta myosin heavy chain mutation in exon 13, a putative defect for HCM is present in only one of 39 affected families. Circulation 1991; (Suppl II) 84(4):II-254. Cardiomyopathy. J Am Coll Cardiol 1992; 19:272A.
• Mares A, Marian AJ, Yu Q-T, Czernuszewicz GZ, Tapscott T, Kelly D, Towbin J, Perryman MB, Roberts R: Genetic screening of 75 families with hypertrophic cardiomyopathy show missense mutations the most common genetic defect. Circulation 1992; 86:1-228.
• Marian AJ, Kelly D, Mares A, Tapscott T, Yu Q-T, Gooch G, Hill R, Towbin J, Perryman MB, Roberts R: The missense mutation in exon 13 of the B-myosin heavy chain gene, a genetic marker for sudden death in patients with hypertrophic cardiomyopathy. Circulation 1992; 861-16.
• Roberts R, Marian AJ, Yu Q-T, Bachinski LL, Mares A: molecular basis of HCM and its implications for cardiac pathophysiology. J Cell Biochem 1993;17D;187.
• Mares A, Greve G, Tapscott T, Roberts R: Screening and identification of known and novel mutations in hypertrophic cardiomyopathy based on molecular scanning with chemical cleavage. Circulation 1993; Suppl 88(4):I-572.
• Greve G, Mares A, Bachinski LL, Roberts R: A sporadic mutation in the B-myosin heavy chain gene transmits hypertrophic cardiomyopathy to the offspring of two generations. Circulation 1993; Suppl 88(4): I-573.
• Belfort M, Mares A, Saade G, Wen T, Rokey R: A re-evaluation of the indications for pulmonary artery catheters in obstetrics: The role of 2D echocardiography and doppler ultrasound. Am J Gyn 1996.
• Gangopadhyay S, Cacela D, Beleskin B, Nagueh S, Lakkis N, Kopelen H, Mares A, Reyes G, Ostojic MC, Zoghbi WA: The Addition of atropine improves the sensitivity of adenosine echocardiography for the detection of coronary artery disease. Circulation 1997; Suppl 96(8):I276.